bartonella bacteria Lyme co-infections

Bartonella henselae is a bacterium that was identified just in 1992, and only in 1997 recognized as a bacterium that causes cat’s claw disease.For examplethequintanaform, causes root fever, but have in mind that there are about 30 different forms of bartonella. You can get infected with this bacteriaby flies, mosquitoes, spiders, cats, dogs – in fact, everything that sticks, bites or scrapes you. It can be found mainly in homeless people, alcoholics but also in people who had a blood transfusion.

In Germany, for 270 healthy people, 30% carry Bartonellehenselae, in Greece for 500 people 20% is its carrier – but I wonder how many infections have not been detected because it is difficult to “grow” in laboratory conditions, hence not many studies have been connected with this bacterium.

Bartonella is a gram negative bacteria living inside cells, which has a double cell wall – internal and external and between them there is periplasmic space. Antibiotics usually pass through one cell wall without penetrating the other, hence the bacteria are so resistant to them. The next line of defense of this bacterium are beta-lactamase enzymes that neutralize beta-lactamase antibiotics. In addition, they have their internal efflux pumps thanks to which they quickly get rid of antibiotic compounds. Normally, if the antibiotic breaks through the first cell wall – it stays for some time in the periplasmic space where it will be pumped out by the efflux pumps.

Bartonella proliferatesevery 21 hours and stimulates the growth of endothelial cells for this purpose. The species of bartonella bacteria adapts to the environment in which it lives and thanks to its genetic material is able to survive attacks of the immune system – it does not exhibit such properties in vitro in the laboratory –however, in vivo does. One of the external proteins found on the bartonella cell wall is the BadA protein, which is a specific camouflage of bacteria against the cells of the immune system. Henselae modifies its outer wall so that it is able to infect red blood cells.

Common symptoms of active bartonella henselae infection include lymph node lymphomas and lymph node inflammation especially at the scratch / bite site. The lymph swelling itself can be really big and persist for a few months. Other symptoms of the disease are naturally a fever of unknown origin, bacterial liver abscess, severe fatigue, muscle pain, reactive arthritis, Kikuchi disease, osteomyelitis, multiple sclerosis, erythema nodosum, skin changes, endocarditis, encephalopathy, headaches, ataxia, memory loss, paraesthesia, aseptic meningitis, meningitis, dementia, mental symptoms including murder, rage, anxiety, severe depression, mental fog, difficult thinking, articulation problem and schizophrenia. Bone pain can be strong and occur even in the foot.

Bartonella Quintana, for example, causes endocarditis, fever, lymphadenopathy, shoulder angiomatosis (movement of the elbow and shoulder), infection of the central nervous system, fatigue, leg pain, shortness of breath, dizziness, hip pain, tremor, abdominal pain, diarrhea, constipation , anorexia, anxiety, insomnia, profuse sweating, bone pain, especially in the tibia, spleen inflammation, breathlessness, i.e. breathing problems, palpitations, and abnormal heart activity. Increased air humidity deepens all pain. In the literature you can also find side effects such as blurred vision, enlarged spleen, abdominal pain or pain in the lumbar region.

Other types of bartonella bacteria include elizabethae, clarridgeiae, koehler, vinsonii, washoensis, melphagi, grahamii, doshiae, alsatica, rochalimae, volans, durdenii, bacilliformis, clarridgeiae, tamiae – practically all cause a number of symptoms similar to the above mentioned.

Recent research shows that bartonella contributes to specific eye problems such as redness of the eye, sensitization to the light, feeling of something under the eyelid, increased production of tears, blurred vision, conjunctivitis, optic neuritis, retinal damage, uveitis, vascular changes , retinal detachment, choroidal choritis, abscesses, retinal hemorrhage, sudden loss of vision, optic neuritis, blurred vision of colors, impaired vision contrast.

Ear symptoms: otitis media, auditory hallucinations, labyrinth disorders.

Neurological problems: facial nerve palsy, coma, demyelination, spinal cord inflammation, neuralgia, Guillain-Barrea syndrome, paresthesia, polyneuropathy, motor impairment, psychosis, hallucinations, transient paresis, muscle spasms, changes in the white matter of the brain. Bartonella imitates multiple sclerosis or bipolar disorders, also many cases of these diseases are incorrect diagnostics.The urological system: glomerulonephritis, urogenital pain, difficulties in emptying the bladder, in the case of glomerulonephritis, blood usually appears in the urine.

Skin symptoms: urticaria, rashes, skin damage, skin ulcers that do not go away and others.

Symptoms associated with conceiving a child: urogenous infections, infertility, low birth weight of a born child, infections in the offspring, miscarriage, inexplicable death of a newborn immediately after birth.

The bartonella antibodies (IFA – indirect fluorescence assay test) show large distortions because bartonella cross-reacts with EBV, CMV, coxiellaburnetti, gondii toxoplasma, chlamydia spp and streptococcus pyogenes (this is the angina) giving false positive results.

Bartonella has very low endotoxicity and vascular endothelial cells have low tolerance to endotoxins. For example, Salmonella exhibits at least 1000 times more toxicity than bartonella bacteria, also herxheimer’s reactions in the case of bartonella do not occur – they are caused by either a different bacterium or are confused with side effects e.g. taking antibiotics.

Bartonella creates biofilms in vitro, in vivo don’t, so things that break down biofilms are not necessary. However, they form vacuoles – they are peculiar balloons filled with bartonella bacteria, macrophages and other cells. Wakuole have a membrane thanks to which they protect the organisms contained in it and are always created when the bartonella bacteria accumulate in a given place.

The way Bartonella works and permeates

After getting into the body, the first thing it takes is erythrocytes, spleen, liver and bone marrow. After some time, the spleen and liver may become inflamed and as the multiplication of bacteria is about 24h.Symptoms will appear between 12-62 days after the infection and will resemble the flu. Problems with bartonella appear when the immune system is in a state of immunosuppression (in other words weakened).
The immunosuppression of the system occurs, for example, in very high stress (for example), in the case of taking, for example, immunosuppressive drugs or in the case of malnutrition, etc.

The external cell wall protein of Bartonella is the orthologue of the A Yersini adherence protein – (an ortholog is something that has the same DNA / RNA and has the same function).

After the bartonella penetration into the host’s body, stimulation of the IL-8 cytokine begins. Stimulation of this cytokine causes the transfer of CD34 + cells to the location of the infection / bacteria. Bartonella penetrates these cells and becomes invulnerable to the human immune system. The infection spreads throughout the body. CD34 + cells are stem cells of the so-called formation cells in the bone marrow, that is why it is mostly infected with this localization in the body. Thanks to their stay in CD34 + cells, they get into every corner of the body where inflammation is going on and where the cells are needed – naturally, the bacteria uses this system for its own purposes – to settle / adhere to other tissues / cells.

The cytokine IL-8 causes an immediate decrease in neutrophils in the blood favors the spread of the above bacteria. After some time, however, levels of IL-8 are increasing, and this is too much to increase the MMP-9 metalloproteinase – it can manifest itself naturally with pain in the joints, but also what one of the ILADS doctors in the US noticed. Streaks / scratches which resemble scratches on the skin made by a cat, which often the people infected have on the skin after a hot shower (increases inflammation).

The biggest problem for the immune system or other methods of combating bacteria in the human body is the fact that this bacterium forms in the CD34 + cells.The already mentioned vacuole in which it lives, and is additionally protected against attacks of various substances or cells through 3 walls that it has to overcome – protective wall of vacuoles and 2 cell walls of bartonella bacteria.

This Bartonella bacterium has a strong affinity for two things – endothelial cells and red blood cells. Endothelium is a thin layer covering all the blood vessels in the body to which bartonella adheres (it can also adhere to cells such as monocytes, macrophages or dendritic cells). The endothelium of blood vessels works on the basis of various mechanisms, being a barrier to substances found in the blood vessels before entering them, regulating the flow of blood or substances from and into the bloodstream. If the endothelium is too porous, the liquid from the blood vessels gets into the body causing swelling. It also controls the movement of leukocytes to and from the bloodstream and is responsible for the narrowing or widening of vessels and inflammatory processes. Endothelial cells also line the heart and endocardium of the heart, causing inflammation there and thereby causing a condition called endocarditis. In addition, the endothelium consists of heparan sulphate (polysaccharide), which is a component of angiogenesis processes (the process of formation of blood vessels).

The bartonella bacterium utilizing heparan increases the formation of blood vessels and endothelium, thus multiplying faster and creates clusters that increase the number of health problems. Naturally, it further inhabits CD34 + cells, stimulates their creation, and continues to expand – it acts like psychopathic Hitler in the times of World War I and II.

bartonella lyme symptoms

Bartonella is also a strong stimulator of vascular endothelial growth factor. It has 2 proteins that are very important in its functioning – they are proteins that adhere bacteria to other cells – BadA protein and FhaB. Thanks to them, it adheres to the surface of the endothelium. On the other hand, the Trw protein allows it to penetrate into the erythrocytes. Unfortunately, this is not the end – thanks to the system called T4SSs (type IV secretion systems) DNA is exchanged with other gram negative bacteria, thanks to which it easily acquires resistance to various toxic compounds (including antibiotics).

Characterized bacterium also stimulates inflammatory transcription factor NFkappaB (it stimulates inflammatory genes), IL-8 inflammatory cytokine, ICAM-1 adhesion molecule and e-selectin (e-selectin increases the permeability of blood vessels which greatly promotes bartonella). As if that was not enough, bartonella has BepC, F, G and A proteins in it that inhibit cell death and stimulate angiogenesis. It also stimulates the inflammatory cytokine IL-1beta, TNF alpha, MMP-2, MMP-9 PI3K, gamma interferons, ERK-1, ERK-2, inducible hypotension factor HIF-1 (this is a factor that activates in states of hypoxia automatically stimulating VEGF factor and increases angiogenesis – notice how people in oxygen masks, which are usedfor special kinds of sports / increase their aerobic capacity and “pump” veins – then these 2 factors are strongly stimulated also such tricks during infection with this bacteria I advise against). E-selectin when it is activated causes neutrophil transfer to the site of infection. Unfortunately, bartonella easily uses them to increase the VEGF factor. Monocytes? the same – they arrive at the place of infection and stimulate the MCP-1 protein, which further transforms them into macrophages and dendritic cells – components of the innate immunity of the immune system. And here, too, the stimulation of VEGF is initiated due to the proximity of monocytes, macrophages and dendritic cells in the presence of bartonella colonies.

Substances that inhibit VEGF cause inhibition of about 50% of the growth of endothelial cells that cause bartonella – inhibition of VEGF therefore inhibits expansion and reduces the number of this bacterium in the host. Bartonella also stimulates the production of protein tyrosine phosphatases (PTPs) – these are enzymes that regulate the growth, multiplication and transformation of cells. They increase, inter alia, VEGF, stimulate the endothelium to grow. PTP blockers, whether herbal or synthetic, will block VEGF and thus bartonella invasions.

ERK1 and ERK-2 kinase as well as PI3K kinase are also stimulated by bartonella – enzymes responsible for cell growth, proliferation, differentiation and their viability. They play an important role in angiogenesis, which is stimulated by the bacterium. ERK-1, ERK-2 and PI3K blockers inhibit endothelial damage, hyperplasia (enlargement of organs or tissue due to excess cell accumulation) and side effects caused by excessive level of cytokine IL-8 .

Bartonella stimulates the production of SOD peroxide dismutase (it would seem to be very good, because all in all the SOD (Cu-Zn) fights free radicals). During infection, leukocytes generate free radicals to kill bacteria because they are not very conducive to any living organism. However, the over-stimulation of the SOD enzyme very easily fights free radicals, neutralizing them so Bartonella protects itself from their attacks and their production from white blood cells. During Bartonella’s skin damage, molecule Pap31 thankfully attaches itself to fibronectin and collagen, which can be inhibited by heparin and thus reduce the infection. Metalopreinases, on the other hand, which bartonella increases without problems, damage not only collagen but also myelin of nerve cells in the brain, which can easily look like and be mistaken for multiple sclerosis. Hippocampus infection will cause problems with memory, uncontrolled aggression, including suicidal thoughts or confusion. Later on, the infection of the cerebral cortex causes memory problems, lack of concentration, problems with speaking and clarity of thought. Hippocampus infection also means problems with body temperature, hunger pangs, increased fatigue, problems with sleep cycles (short / long phase). For example, the infection of the spinal cord or brain can be transformed into chronic migraines or deterioration of cognitive functions.

Bartonella, by entering the erythrocytes, deprives them of nutrients, including iron, which he needs. In erythrocytes there is about 2.5 grams of iron – about 65% of all iron in the body. Low levels of CD4 + cells allow faster spread of infection (making it very easy to start a contagion) – bartonella alone causes a decrease in these cells.

As the following information was collected and developed about 10 years ago, below are the newest (and the little older) scientific materials about different varieties of bartonella bacteria including various interesting facts related to this infection.

  • Quintana can be found in demodex and lice.
  • Hypogeneity, swelling, erythema, cyanosis of both feet – these are the symptoms of Bartonella koehlerae and vinsonii, which were diagnosed at one veterinarian.
  • Henselae can cause the so called floater.
  • Henselae may cause inflammation of the liver.
  • May cause inflammation of the spleen.
  • Causes inflammation of the thyroid (Henselae).
  • In cats, it causes hyperglobulinaemia (increased levels of immunoglobulins in response to infection).
  • It may relate tocataract.
  • Quintana causes glomerulonephritis and kidney damage.
  • Causes disorders of cognitive functions (henselae and vinsonii).
  • Quintana causes optic neuritis.
  • Bartonella generally inhibits apoptosis (cell death and thus promotes the formation of tumors) and stimulates hypoxia (i.e. increases the hypoxic state which affects the increased activity of new blood vessel formation).
  • B.Koehlerae causes fears, depressions, mood behaviors, severe headaches, muscle spasms, stiff joints, decreased vision, decreased tactile sensitivity or hallucinations.
  • Bartonella has a BadA protein on its surface that allows it to adhere to cells (henselae variety), while Quintana has a Vomps protein.
  • Bartonella causes Still’s disease.
  • Unexplained cases of Epilepsy – this may be Bartonella henselae
  • Causes inflammation of the retina and inflammation of the blood vessels.
  • If there is inflammation of the bones and bone marrow with inflammation of the lymph nodes, you can suspect Bartonella henselae.
  • Causes IgA nephropathy (leads to glomerulonephritis).Haematuriamay appear.
  • May cause hypercalcemia (increased calcium in the blood) through increased production of d3 1.25OH meafolite (from time to time such cases can be observed).
  • Of the 18 tested antibiotics, only 4 have bartonella effects – rifampin, ciprofloxacin (which side effects can be read in the medical literature, long articles), intramuscular gentamicin and trimethoprim-sulfamethoxazole.
  • Bartonella h. Increases levels of IL-2, IL-6 cytokines (inflammatory cytokines) and anti-inflammatory cytokine IL-10.
  • Bartnella h. Causes retinal detachment.
  • Bacillary angiomatosis causes Bartonella h. And q. (These are red / purple bumps or papules that may be ulcerated). They are very similar to lichen. They often arise in the case of a weakened immune system.
  • Bartonella h. Can mimic the symptoms of lupus.
  • It probably breaks down the spinal disc (Bartonella h.)
  • Causes bone marrow inflammation.
  • Copper and iron protect against anemia caused by Bartonellemuris.
  • Mimics the symptoms of multiple sclerosis.
  • Stimulates angiogenesis, which is a very unfavorable phenomenon in the case of cancer or AMD vision problems.
  • The BepA protein found in B.henselae and quintana causes suppression of endothelial cell apoptosis while contributing to tumors.
  • It can lead to retinal tumors.
  • Bartonella proteus reduces the activity of the immune system, paralyzes red blood cells and occupies the brain.
  • It is not a frequent phenomenon but Bartonella henselaecan also cause Guillain-Barré syndrome.
  • Bartonella henselae may damage the duodenum.
  • B.grahamii increases the levels of interferon alpha and beta (the same, moreover, does b.henselae, however, to a lesser extent). Strong activation of the above interferons inhibits the proliferation (division / proliferation) of B lymphocytes necessary for the production of antibodies and thus the detection of pathogens. Problems with lymph are dependent on the above interferons.
  • Thanks to the TrwJ1 and TrwJ2 proteins, Bartonella birtlessi attaches itself to erythrocytes.
  • B.vinsonii and henselae stimulates HIF-1alpha (it is a stimulant of VEGF – it causes hypoxia and increases angiogenesis). They also stimulate the VEGF factor directly.
  • Bartonella vinsonii may cause cyanosis of the feet and the appearance of erythema.
  • Both Chlamydia and Bartonella have antigens that cause cross-reactions when detecting these infections.
  • B.vinsonii increases the ratio of CD4 + / CD8 + T lymphocytes if it is additionally an ehrlichiacanis infection.
  • B.henselae causes liver granuloma.
  • Bartonella henselae causes allergic purpura (Henoch-Schönlein purpura).
  • Bartonella henselae causes juvenile arthritis.
  • Red blood cells of alcoholics contain larger amounts of bartonellaQuintana than people who do not drink alcohol.
  • Causes a purple area of ​​the skin that suggests damage to the blood vessels in the upper and lower areas.
  • Bartonella quintana inhibits the signal path of p38 MAPK and SAPK / JNK which causes cell death (also inhibits cell death) and thereby strengthens the formation of cancerous tissue. It increases the activity of bcl-2 kinase, which is responsible for inhibiting cell death, which also does not favor the inhibiting the proliferation of cancerous tissue.
  • Bartonella h. Causes the inflammation of the hip intestine (the last section of the small intestine before joining with the large intestine) – it is simply Corohna’s disease (mostly).
  • People can be infected with Bartonella without any problems by a blood transfusion.
  • Increases the levels of inflammatory transcription factor NFkappaB.
  • In cats, stimulates the cytokine IL-4 (anti-inflammatory cytokine).
  • B.Quintana increases the IL-8 inflammatory cytokine.
  • B.henselae causes an increase in ANCA antibodies
  • One of the b.henselae proteins during the examination of the presence of infection in the body shows cross-reactions with the Brucella melitensis proteins, Mycoplasma pneumoniae, Francisellatularensis, Coxiella burnetii and Rickettsia typhi.
  • B.quintana has the HbpA protein in it which is its virulence factor (inhibition of this protein will inevitably affect the lack or minimization of infection of these bacteria).
  • Bartonella Quintana increases the levels of IL-2, IL-1beta, IL-6 and IL-10 (which increases due to inflammation which it tries to inhibit – IL-10 is an anti-inflammatory cytokine). Excessively elevated
    IL-10 causes inflammation.
  • Bartonella was detected in humans in 1889 and classified as a cat-claw disease. about 10% of infected people have eye problems associated with this infection.
  • Bartonella quintana causes endocarditis (causing inflammation of the aorta).
  • Bartonella henselae sometimes causes pain in the lower part of the spine (it is probably related to the inflammatory conditions it causes).
  • Cats are the most frequent carrier and organism carrying and transferring B.henselae on people
  • One of the proteins found on Bartonella quintana is very close to the yersini protein – it is about the YadA protein – I would pay attention to it when viewing laboratory results. Generally, this is the virulence protein of Yersinia enterocolitica and in the case of both bacteria allows the bacteria to adhere to the tissues / cells of the human body (especially to type IV collagen).
  • Attacks endothelial cells creating inflammation and tumor formation in them.
  • Henselae causes retinal neuritis and follicular conjunctivitis.
  • Blurred (blurred vision) and headaches are one of the symptoms of bartonella infection (even without fever and lymphodenomapti or enlarged lymph nodes).
  • Henselae causes a decrease in CD4 + cells. Elevated TNF alpha and gamma interferons are a tool for the immune system to fight this type of bartonella.
  • The high probability of Bartonella infection occurs in people infected with HIV (probably it is low CD4 + cells).
  • The case of a man with Bartonella who caused endocardial prostheticroot inflammation and a young boy’s case.
  • Quintana can cause Guillain-Barr syndrome and also contributes to encephalopathy. In the second study, the case of a girl infected with Bartonella but without her typical symptoms outside the GB syndrome.
  • Quintana increases VEGF. Anyway henselaedoes also, plus NFkappaB.
  • Rifampin (colloquially called rifa among healers, is an antibiotic that is already resistant to bartonella quintana strains.
  • Gentamicin is the antibiotic that Bartonella Henselae is resistant to.
  • Abdominal lymphoma is very often an infection with Bartonella.
  • Bartonella h. May cause a spleen rupture.
  • It is very possible that Bartonella henselae contributes to myeloid leukemia.
  • Bartonella henselae causes lateral inflammation of the spinal cord.
  • Henselae causes osteomyelitis.
  • Henselae causes blurry eye / eyes, may cause glaucoma.
  • ialA is a Bartonella bacilliformis gene that allows it to penetrate erythrocytes (red blood cells).
  • Bartonella henselaeoveractivates the MCP-1 gene, which has a number of important functions in the body, but at the same time its increased level correlates with the existence of inflammation of the joints. The functions of this gene are as follows: luring monocytes and T-lymphocytes, eosinophils, mast cells and basophils, and additionally stimulating cytotoxic destruction of cells by CD8 + lymphocytes and NK lymphocytes. This is particularly important in the course of cancer. In addition, it affects the release of histamine and synthesis of leukotrienes by mast cells and basophils.
  • Bartonella increases the activity of the Bax and Bcl-2 genes, resulting in a lack of cellular apoptosis – it is very dangerous in the case of any type of cancer. Increases IL-8 inflammatory cytokine in hepatocytes, monocytes and endothelial cells.
  • Bartonella henselae causes glomerulonephritis.
  • Bartonella henselae stimulates the production of interferon gamma (activates NK cells and macrophages), however, during this activation, the level of IL-10 increases and in some people the interferon gamma may be very high, and in others definitely too low.
  • Bartonella causes leukocytosis, thrombocytosis, hypercalcemia and elevated angiotensin-converting enzyme. Inflammation of the liver and spleen – this infection is normal.
  • B.quintana causes an increase in inflammatory cytokines by stimulation of TLR2
  • B. bacilloformis may be resistant to ciprofloxacin.
  • People with a positive HLA-B27 antigen (tested for suspected autoimmune disease) are more likely to be seropositive when it comes to infection with Bartonella.
  • In the case of a low level of interferon gamma, it should be stimulated because it can contribute to getting rid of Bartonella henselae infection (interferon gamma stimulates macrophages).

herbs bartonella

What is highly recommended by Stephen Buhner – one of the best American herbalists, a pioneer in the techniques of natural borreliosis and co-infection treatment.

Reduction of inflammation through inhibition of the transcription factor NFkappaB – bidenspilosa, Chinese senega root, Chinese Baychea thyroid, Cordyceps, EGCG, celandine herb, houttuynia, Japanese knotweed, kudzu, luteolin, olive oil,

pomegranate juice, Chinese senega (root) and schisandrasinensis (Chinese citrus),

Inhibition of the IL-8 inflammatory cytokine – cordyceps, EGCG, isatis, knotweed, NAC, grenade

Inhibition of EGF – berberine, EGCG, ginkgo biloba, quercitin

Inhibition of e-selectin – ginkgo biloba, knotweed, kudzu, milk thistle, schisandrasinensis

Inhibition of PTP: elder (Aegopodiumpodagraria L.) and hawthorn (Crataegusoxyacantha)

ERK inhibition – Baikal thyroid, cordyceps, EGCG, celandine herb celandine, knotweed, kudzu, olive oil

Organ protection against bartonella is provided bysidaacuta (spleen), hawthorn (Crataegus) (heart), milk thistle (liver) and red root (ceanothus) (lymph), endothelium, knotweed, l-arginine and EGCG. Red blood cells, in turn, will protect

NAC, bidens, alchornea and cryptolepis. Support for CD4 + cells – Rhodiola and ashwagandha (also reduce inflammation).

Typically, houttuynia,alchorneacordifolia and isatiswill be antibacterial in this case, as well as the aforementioned sidaacuta.

His basic protocol for Bartonella:

Japanese knotweed: tincture, 1/4 to 1/2 teaspoon 3x a day
EGCG with quercitin: 800 mg EGCG plus 1,200 mg quercitin per day.
L-arginine: 500-1,000 mg 3x a day (do not use in case of active infection with herpes / gill virus)
Cordyceps: tincture, 1/4 teaspoon 3x a day or powder – 1 teaspoon 3x a day or capsules 2grams a day 3x a day.
Sidaacuta: 30-60 drops of tincture 3-4x a day.
Red root: tincture 1/4 to 1/2 teaspoon 3x a day
Milk thistle: Extract, 1200mg per day
Hawthorn (Crataegusoxyacantha): Tincture, 1/4 to 1/2 teaspoon 3x daily
Rhodiola / ashwagandha combination of these 2 herbs (fifty-fifty): Tincture, 1/2 teaspoon 3x daily.
Isatis / houttuynia / alchornea combination of these 3 herbs, equally each: Tincture, 1/2 teaspoon 3x a day
Pomegranate juice throughout the day.
Vitamin E (alpha-tocopherol): 200 IU or 150 mg daily (optional).

Depending on the symptoms, add to the basic protocol
If there are problems with the nervous system / brain

Baikal thyroid, tincture, 1/4 tablespoons 3x a day
Celandine celandine, tincture 1/4 teaspoon 3x a day
Kudzu root, tincture 1/4 teaspoon 3-4x a day
N-acetylcysteine ​​2,000 mg 2x a day
Common celandine, tincture 1/4 to 1/2 teaspoon to 6x a day

If there is a problem with reduced blood flow in the brain (something for people with multiple sclerosis)

  1. Ginkgo biloba, 1/4 teaspoons tincture 3x a day

Neurological pain

  1. Celandine celandine tincture 1/4 teaspoon 3x a day
    2. Kudzu root tincture, 1/2 teaspoon 3-4x a day

If there is a problem with hyperactivity / hysteria

  1. Pulsatilla (Pasque flower), tincture, use 10 drops until the symptoms pass
    2. Motherwort tincture, 1/4 to 1/2 teaspoon to 6x a day
    3. Coral root tincture, 30 drops to 6x a day
    4. Baikal thyroid, tincture 1/4 teaspoon 3x a day

If there is panic

  1. Baikal thyroid, 1/4 teaspoon tincture 3x daily
    2. Verbena tincture, 30 drops 6x a day

If there are problems with sleeping

  1. Melatonin
    2. Ashwagandha tincture one hour before bedtime or in powder form or in a capsule 1gram an hour before bedtime
    3. Baikal thyroid, tincture 1/4 teaspoon 3x a day
    4. Motherwort, tincture 8 ml at bedtime if melatonin does not help

If tired, use for 6 months

  1. Siberian Ginseng, tincture 1/4 teaspoon every day in the morning
    2. Rhodiola tincture, 1/4 teaspoon 3x a day
    3. Schisandra sinensis tincture, 1/4 teaspoon 3x a day
    4. The cordon, tincture 1/4 teaspoon 3x a day

In the case of continuous cough:

1.Bidens pilosa tincture, 1/4 teaspoon 3-6 times a day

In case of anemia and problems with erythrocytes

  1. Sidaacuta tincture, 1/2 teaspoon tincture 3-6x a day
    2. N-acetylcysteine, 4,000 mg twice a day
    3. Bidenspilosa tincture, 1/2 teaspoon 6x a day

In the case of excessive weight loss

  1. Shiitake Mushroom, 6-16 grams per day

In order to improve the detoxification and to neutralize herxheimer reactions

  1. Zeolite: 15 drops 3-4x a day or powder 2 teaspoons or 3 large capsules 3x a day

What does Byron Whiterecommend– doctor / herbalist recommended by Dr.Horowitz (this is the content of A-Bart tincture made from the following herbs / plants for 40% alcohol)
Allum sativum (tuber) (Garlic), unacariarhynchophylla (Gou Teng) (stems and spikes), glycyrrhiza glabra (root) (Liquorice), azadirachtaindica (Indian lilac) (leaf), citrus paradisi (seeds)(Grapefruit) , hemidesmus indicus (root), syzygiumaromaticum (bud) (Carnation – buds), usneabarbata (lichen), Phytolaccaamericana (root) (American Scarlet)




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